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Interesting tests were carried out on mice and amazing results were found. Keep reading to know more about this interesting research!
The latest researches have changed the traditional belief that insufficiency of Serotonin (a brain chemical that serves the purpose of a messenger) plays a fundamental role in creating behavioral changes like depression in human beings.
The researchers of the ACS Neuroscience Chemical Journal states that the experiments on the brains of mice that were ineligible to produce serotonin and that were traditionally marked as the depressed ones didn’t display any symptoms that prove they were in a depression.
The students, lead by Donald Kuhn, of Wayne State University School of Medicine and John D. Dingell VA Medical Center observed that depression is the notified fundamental dilemma amongst the general public health issues.
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The popular medicine of depression Prozac that was introduced in the latter part of the 1980s works basically by raising its quantity of Serotonin in the human brain.
This was the reason that researchers tend to trust that the fundamental solution of depression is by increasing the levels of Serotonin in the brain.
Similarly, many other medicines to take care of depression came into being that was all relying on the same basic idea.
However, by this time, scientists are well aware that the majority of people (60-70%) of people keep on feeling depressed, despite using these medicines.
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To carry out this, they made “Unconscious” mice whose brains were in short of the capability to generate serotonin. After that, they performed various behavioral examinations on them.
Amusingly, the “Unconscious” mice were obsessive and tremendously violent, but they didn’t display symptoms of depression in them. One more amazing discovery is that the “Unconscious” mice acted in a similar manner majority of normal conscious mice did, at times they were put under strain.
The scientists declared that the above discoveries are perhaps the proofs that alone serotonin isn’t the fundamental cause of depression and several other causes are certainly concerned.
To sum up, they proposed that these findings may radically change as the research for fresh antidepressant medicines undergo in the upcoming time.
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